^Araki T، Milbrandt J (أكتوبر 1996). "Ninjurin, a novel adhesion molecule, is induced by nerve injury and promotes axonal growth". Neuron. ج. 17 ع. 2: 353–61. DOI:10.1016/S0896-6273(00)80166-X. PMID:8780658.
قراءة متعمقة
Guimarães PE، Fridman C، Gregório SP، وآخرون (2007). "WITHDRAWN: Association between polymorphisms in genes of inflammatory response and axonal repair with spinal cord injury". Spinal Cord. DOI:10.1038/sj.sc.3102122. PMID:17893696.
Cardoso CC، Martinez AN، Guimarães PE، وآخرون (2007). "Ninjurin 1 asp110ala single nucleotide polymorphism is associated with protection in leprosy nerve damage". J. Neuroimmunol. ج. 190 ع. 1–2: 131–8. DOI:10.1016/j.jneuroim.2007.07.015. PMID:17825431.
Chadwick BP، Heath SK، Williamson J، وآخرون (1998). "The human homologue of the ninjurin gene maps to the candidate region of hereditary sensory neuropathy type I (HSNI)". Genomics. ج. 47 ع. 1: 58–63. DOI:10.1006/geno.1997.5084. PMID:9465296.
Araki T، Zimonjic DB، Popescu NC، Milbrandt J (1997). "Mechanism of homophilic binding mediated by ninjurin, a novel widely expressed adhesion molecule". J. Biol. Chem. ج. 272 ع. 34: 21373–80. DOI:10.1074/jbc.272.34.21373. PMID:9261151.{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link)